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The anabolic data come from rodents, often in artificial catabolic states, using administration routes and durations designed to probe pharmacology rather than model therapeutic use

Studies suggest a role for brain-derived GLP-1 in the control of eating and reward (80, 81), but there is no evidence that peripherally administered GLP-1RAs can directly access GLP-1Rs in these regions, as demonstrated by the lack of fluorescently labeled semaglutide in these brain nuclei

As expected, in our study nAngptl4-mediated inhibition of LPL activity only depleted half of the total lipase activity in typical human PHP, with the nAngptl4-resistant lipase activity most likely being HL, and to a much lesser extent endothelial lipase (EL) (Figure 3B)

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