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All the experiments in this study were approved by and performed in accordance with the Animal Ethics Committee, Lund and Malm, Sweden (Ethical permit number M458-12)

Mitochondria-dependent ferroptosis plays a pivotal role in doxorubicin cardiotoxicity

[66] first revealed that LONP1 is a critical anchoring protein for MAM in cardiomyocytes, with its absence leading to reduced MAM formation and mitochondrial fragmentation

The intervention was delivered as single weekly intravenous infusion, with each dose administered over 15 min in 50 mL of saline, for two consecutive 8-week periods, separated by a 1-week washout