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These findings suggest that TGF- may suppress SC characteristics by preventing c-MET overactivation in subgroups of gliomas (Fig

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Pulmonary pathological changes and underlying mechanisms Due to systemic and pulmonary immunoinflammatory and immunothrombosis during ARDS, as well as subsequent disruption of the alveolar-capillary barrier, various cellular pathological changes, including various types of cell death, cellular senescence, and cellular dysfunction, have been demonstrated to occur in damaged pulmonary tissue