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The first studies with BPC 157 focused on its prominent beneficial effects on gastric and intestinal injuries induced by diverse ulcerogens (18)

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however, starting from the age of 3, progressive myopathy was developed, and, at the age of 8, a homozygous nonsense mutation in the MPTb domain of FLAD1 was found, which produced a shortened protein that retained some level of FADS activity [75]