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This signature exhibited an overrepresentation of mediators of mitochondrial metabolism (Fig

Repeated stimulation likely downregulated Trpa1 , a common adaptive mechanism to sustained receptor activation 73 that may modulate the excitability and peptide turnover of EWcp/UCN1/CART neurons, potentially influencing alcohol consumption pattern

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In summary, SLC7A5 overexpression was able to reverse the protective effects of YTHDF1 knockdown, revealing the critical role of SLC7A5 in the YTHDF1-mediated cytoprotective mechanism