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Mechanistically, NNMT suppression increased adipose SAM and NAD, upregulated polyamine flux enzymes (ODC, SSAT), and altered histone methylation consistent with a shift toward oxidative metabolism [2]

Moreover, oxidative stress be a fundamental pathophysiological mechanism in different inborn metabolic errors, further emphasizing the role of oxidative stress in cobalamin-related disorders [119]

Its ability to selectively target senescent cells has positioned it at the forefront of scientific investigations involving aging, cellular rejuvenation, and tissue regeneration

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