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Mutant p53 can influence the remodeling of the extracellular matrix (ECM) and the expression of cell adhesion molecules, thereby increasing the invasiveness of tumor cells

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For example, the multifunctional nano-epigenetic inhibitor CREDIT composed of polymeric polylactic acid-glycolic acid (PLGA) grafted with histidine (PLGA-his) loaded with the epigenetic inhibitor TMP195, modified with red blood cell membranes and conjugated with the TAM-targeting peptide CRV, can reprogram M2-type TAMs into the M1 phenotype, reduce MDSCs, increase the number of T cells in tumors and inhibit tumor growth (215)

In individuals with IBD, proinflammatory cytokines downregulate OCTN2 through the PPAR/RXR pathway, resulting in reduced carnitine uptake by intestinal epithelial cells that leads to the dysfunction of intestinal regeneration and repair and further deterioration of IBD [232]