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Several clinical trials have demonstrated their capacity to improve glycemic control, lipid profiles, and inflammatory markers in patients with T2D (Zhang et al., 2025)

It also reduces the alanine aminotransferase and aspartate aminotransferase levels and suppressed the apoptosis in hepatocytes (4,16)

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however, its exact contribution to brain Se transport was not systematically studied and it seems to be rather limited since megalin / mice do not exhibit neurological phenotype associated with Se deficiency (Kurokawa et al., 2014b), typical for Selenop / or Lrp8 / mice