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While there is evidence of disrupted serotonergic neurotransmission in AD (267), the role of neurosteroids, such as DHEAS in the progression of this age-related neurodegenerative disease has not been fully elucidated
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The findings revealed that BPC-157 accelerated the outgrowth of tendon fibroblasts, enhanced the survival of these cells under H2O2-induced stress, and promoted the migration and spreading of tendon fibroblasts in a dose-dependent manner
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