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The knob mutation (T366Y) was introduced into pFUSE-HA(PR8)-hIgG1-Fc2, while the hole mutation (Y407T) was introduced into the pFUSE-naked-hIgG1-Fc construct either alone or in combination with the REW substitutions

These effects (i.e., rapidly reestablished reorganized blood flow) attenuated/eliminated the lesions in the brain (intracerebral and intraventricular hemorrhage), heart (congestion, myocardial infarction, severe arrhythmias), lung (hemorrhage), and congestion and lesions in the liver, kidney, and gastrointestinal tract [54]

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