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In multiple sclerosis (MS), a neuroinflammatory disease driven by immune-mediated destruction of myelin-producing oligodendrocytes, CYP51A1 has emerged as a potential therapeutic target

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In a severe acute pancreatitis rat model induced by sodium taurocholate solution, catalpol demonstrates therapeutic effects on intestinal inflammation by downregulating the HMGB1/RAGE signaling pathway ( As a promising candidate for demyelinating diseases, catalpol promotes oligodendrocyte generation and myelin repair, alleviates clinical neurological dysfunction, inhibits inflammatory infiltration, and increases the proportion of Treg cells (Wu M