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This altered environment exerts selective pressure, reshaping the microbial community by reducing beneficial commensal bacteria such as Bifidobacterium and Lactobacillus and promoting opportunistic pathogens, especially within the phylum Proteobacteria (78, 79)

Some investigations have demonstrated that the two compounds, acting as agonists of the GSH antioxidant system, well prevent oxidative stress and reduce heart damage in some animal cardiomyopathy models, for instance, the ISO-induced myocardium infarction model [159, 160]

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