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Length-independent telomere damage drives post-mitotic cardiomyocyte senescence

Loren Pickart first identified GHK in 1973 when he observed that it caused older human liver tissue to produce proteins similar to those found in younger tissue

demonstrated that GSSH efflux occurs proximal upstream of NLRP3 inflammasome activation after exposure to DAMPs, the result being ROS accumulation and redox imbalance, which collectively trigger inflammasome activation

The image provides a comprehensive overview of the key cellular and molecular players involved in the pathogenesis of RA, illustrating the complex interactions between different cell types and various inflammatory mediators that contribute to the development and progression of the disease