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Extracellular cysteine/cystine redox potential controls lung fibroblast proliferation and matrix expression through upregulation of transforming growth factor-beta

These receptors are expressed in a wide variety of tissues including the gastrointestinal tract, central nervous system (specifically cells of the tuberomammillary nucleus of the hypothalamus), both airway and vascular smooth muscle cells, endothelial cells, chondrocytes, monocytes, neutrophils, dendritic cells, T and B lymphocytes, adrenal medulla, and the cardiovascular and genitourinary systems

Oncogenic mutations in KRAS cause a shift of equilibrium from the inactive guanosine diphosphate (GDP)-bound state to the active guanosine triphosphate (GTP)-bound state, thereby amplifying proliferative signaling pathways and promoting uncontrolled cell growth 2,3,4