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This rebound occurs because GLP-1 therapy reduces your metabolic set pointthe weight your body 'defends'but stopping the medication doesn't permanently reset that point

Notably, this effect was independent of glucose-dependent membrane depolarization, as increased GLP-1 release in response to siRNA knock-down of the essential NMDA receptor subunit GluN1 was present in both basal Mg 2+ -free buffer conditions and buffer containing glucose

This dual oversight allows both teams to monitor liver function tests, metabolic markers, and gastrointestinal tolerance simultaneously rather than in silos

Residual confounding possible