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The system gets flooded with NAD+ at levels up to ten times higher than normal, which raises concerns about: Potential mitochondrial dysfunction Possible sirtuin overactivation Disruption of methylation processes [8] NAD+ at very high levels may block poly(ADP-ribose) polymerases (PARPs) and possibly compromise DNA repair mechanisms [8]

Moreover, they influence immune recruitment and migration by altering the synthesis of chemokines and adhesion molecules (271, 276)

575:693698 Bersuker K, Hendricks JM, Li Z, Magtanong L, Ford B, Tang PH, Roberts MA, Tong B et al (2019) The CoQ oxidoreductase FSP1 acts parallel to GPX4 to inhibit ferroptosis