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Three separate anti-inflammatory mechanisms (NF-B via GHK-Cu, growth factor modulation via BPC-157, melanocortin receptors via KPV) beats two

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Studies have shown that thyroid autoimmunity is influenced by genetic variations in adhesion molecules (CD14, CD40), cytokines and their receptors, self-antigens, apoptosis-related gene products, and the promoter region of selenoprotein S (Klubo-Gwiezdzinska And Wartofsky 2022)