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Mechanism of Action at Cellular Level Angiogenic Pathway Activation One of the most extensively characterized mechanisms of BPC-157 involves its pro-angiogenic effects mediated through the vascular endothelial growth factor receptor 2 (VEGFR2) signaling system

Hepatic cytochrome P450 isoenzymes CYP1A2 and CYP3A4 mediate N-de-ethylation, so polymorphisms or competitive inhibition at these pathways (e.g., by certain antidepressants or azole antifungals) may increase systemic exposure, underscoring the importance of individualizing dosage and monitoring serum concentrations in prolonged infusions