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The authors speculated that increased GSH reflected a compensatory response to ongoing inflammatory processes related to brain injury

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J Young Pharm 5:112115 Lee W, VanderVen BC, Fahey RJ, Russell DG (2013) Intracellular Mycobacterium tuberculosis exploits host-derived fatty acids to limit metabolic stress

Over 12 different genetic loci have been associated with congenital hyperinsulinism but the most common genetic cause, which accounts for up to 60% of cases (27), are inactivating mutations in ABCC8 and KCNJ11 , which encode the two subunits of the beta cell K ATP channel