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Paiva, E

Extracellular arginine deprivation enhances venetoclax sensitivity in acute myeloidleukemia via mitochondrial fission impairment and apoptotic priming

This FAK-paxillin signaling axis connects BPC-157's molecular pharmacology to its observed tissue-level effects in a mechanistically coherent way: a compound that enhances endothelial FAK activation will simultaneously promote cell survival (resistance to anoikis), cell migration (capillary sprouting), and cell-matrix adhesion (structural integrity of new vessels)

Gut dysbiosis may thus result in deficiencies of critical micronutrients, notably iodine, selenium, iron, zinc, and vitamin D, which are indispensable for optimal thyroid function and regulation (Knezevic et al., 2020)