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Fascinatingly, exendin-4 appears to stimulate cholangiocyte proliferation through the GLP-1 receptor, hereby preventing cholangiocyte apoptosis in models of bile acid-induced damage and models of ductopenic cholangiopathies (95, 96)

One study found significantly higher fasting endogenous GLP-1 levels in patients with severe OSA compared to those with less severe OSA, potentially to control fasting glucose levels in patients with insulin resistance