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Endothelial injury generated directly by intracellular viral replication and by ACE2 downregulation, exposing cells to angiotensin II in the absence of the modulator effects of angiotensin 17 (270, 271), and vascular chronic inflammation promoting the development of tissue macrophages overloaded by cholesterol (foam cells), both increase the possibility of acquiring a severe COVID-19 infection (170, 209, 258, 259, 272, 273)

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Anderson While non-oxidant doses of intravenous vitamin c (IVC) and glutathione (GSH) are synergistic in support of the ReDox cascade between physiological compartments (Cytosol-Cell Membrane-Plasma-Plasma Lipids)* it was previously theorized that an oxidant therapy such as high dose IVC (HDIVC) may be defeated by addition of GSH in proximity

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