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[8] [9] Activation of GLP-1 receptors stimulates glucose-dependent insulin secretion, suppresses glucagon release, slows gastric emptying, and reduces appetite via central nervous system pathways

This designation allows the use of these compounds strictly for in-vitro laboratory testing and experimentation

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This is in agreement with our recent report that the majority of markers of dysmetabolism manifest early during cardiometabolic disease development 31