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This results in the inactivation of NF-B-dependent inflammatory factors such as TNF- and IL-6, as well as the blockade of cell signaling molecules such as MAP kinases

Schoenenberger 1984

The suppression of antioxidant proteins, glutathione synthesis enzymes, and NADPH-producing enzymes would lead to ROS accumulation in cancer cells and subsequent oxidative damage to various intracellular components, thus compromising the cell viability

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