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42 Triglycerides themselves are not directly toxic to the pancreas, but free fatty acids generated through lipase-mediated hydrolysis can induce pancreatic inflammation, upregulate proinflammatory mediators, impair mitochondrial complex formation in acinar cells, and cause pathological increases in intracellular Ca concentrations

Additionally, DET1 R306 forms a salt bridge with COP1 D688 (Fig