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FIGURE 1 3.2 ATT-I inhibited overproduction of colonic pro-inflammatory cytokines We utilized real-time qPCR to evaluate the transcript profiles of various pro-inflammatory mediators within the colon, aiming to clarify the regulatory role of ATT-I in systemic and mucosal inflammation

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Nrf2 is controlled by two distinct -TrCP recognition motifs in its Neh6 domain, one of which can be modulated by GSK-3 activity

Clock genes such as Rev-erb repress distal enhancers resulting in the repression of macrophages (Lam et al., 2013), whereas Bmal1 which controls rhythmic trafficking of inflammatory monocytes to sites of inflammation (Nguyen et al., 2013)