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To explore the enhanced dependency on mitochondrial function, we further explored the acute metabolic stress imposed by 5FU treatment

doi:10.1039/c2cs35066a

Alternatively, peptides/peptidomimetics could be designed to target the eIF4E-upstream regulators, as the PI3KmTOR pathway, likely inhibiting the interactions of mTORC1 with Raptor and other partners, affecting the activity of the downstream S6K or 4E-BP proteins (Figure 1) (134)