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The impact of drug treatment on GLUT1/6 and SREBP2 expression levels was also evaluated
Firstly, given the evidence that glutathione and glutamate levels in the brain are affected by a risk variant of the gene coding for the glutathione synthesis enzyme, the role of genetic influences should be investigated [22]
Research suggests that it may be more effective than non-modified glutathione
However, under oxidative stress conditions, Nrf2 dissociates from Keap1 and translocates to the nucleus, where it activates various antioxidant genes, including HO-1, protecting cells from oxidative damage
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