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By day 52, cumulative Dox exposure reached 30 mg/kg, at which point significant LV dilation and functional failure are known to occur, consistent with prior models [1]

This study describes the crucial molecular component and physiological relevance of glucose-sensing mechanisms in hypothalamic neurons

Because the study did not include corresponding drug treatment in chow-fed mice, potential high-fat diet-dependent gut microbial compositional effects of liraglutide/GUB09-145 treatment cannot be ruled out

In this review, we provide an mtROS-centered framework to bridge this gap by integrating anatomical and histopathological changes with mitochondrial dysfunction, redox dysregulation, and catabolic signaling in rotator cuff injury-induced supraspinatus atrophy