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doi: 10.1016/j.ebiom.2018.06.005 Summary Keywords tubular injury, diabetic kidney disease, mechanism, therapy, programmed cell death, stem cell Citation Wang Y, Jin M, Cheng CK and Li Q (2023) Tubular injury in diabetic kidney disease: molecular mechanisms and potential therapeutic perspectives
Across these models the recurring theme, where effects are seen, is not that GHK-Cu creates tissue from nothing, but that it appears to influence the normal repair sequence recruitment of fibroblasts, matrix synthesis, cross-linking supported by copper-dependent lysyl oxidase, and angiogenesis
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