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Activation of the CaSR, when there are normal levels of serum calcium, results in increased proteolysis of PTH resulting in little secretion of the hormone from the parathyroid gland

ER Ca 2+ release, sustained store-operated Ca 2+ entry, and persistent cytosolic Ca 2+ elevation can continue to damage PACs even when mitochondrial Ca 2+ entry through MCU is reduced, because calcium-dependent digestive enzyme activation, PM injury, and failure of ATP-dependent ion pumps remain operative

Because these clinical guidelines shift to match emerging medical data, having an honest, proactive conversation with your primary care provider is always your best first step

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