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These effects have been confirmed in animal experiments, but not in humans, and the specific mechanism remains unknown

A gap over 90 days between prescription fills marked the end of treatment, to account for immortal time bias [23]

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Glucose-dependent insulinotropic polypeptide (GIP) stimulation of pancreatic beta-cell survival is dependent upon phosphatidylinositol 3-kinase (PI3K)/protein kinase B (PKB) signaling, inactivation of the forkhead transcription factor Foxo1, and down-regulation of bax expression