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The glucose-dependent mechanism of insulin release distinguishes GLP-1 agonists from many other antidiabetic medications, resulting in a lower risk of hypoglycemia when used as monotherapy

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The high neutrophil to lymphocyte ratio in critically ill patients, the intense neutrophil infiltration in pulmonary capillaries and into pulmonary alveoli, and the increased levels of circulating neutrophil extracellular traps (NETs) clearly show neutrophil involvement/activation favoring intense ROS production and oxidative damage caused by lipid peroxidation, and protein and DNA oxidation (Komaravelli and Casola, 2014