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It sits in the extracellular matrix and, upon injury or mechanical stress, binds to c-met receptors on muscle satellite cells, activating them from quiescence so they can multiply and rebuild muscle fibers

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1 When IGF-1 binds to IGF-1R, the tyrosine kinase activates the phosphoinositide 3-kinase (PI3K)-AKT pathway and many integral proteins involved in cellular metabolism, apoptosis, cell adhesion, and angiogenesis are regulated within this pathway

Statistical methods were not used to chose sample sizes