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In vitro experiments further confirmed that Que significantly upregulated AMPK expression in oleic acid (OA)-stimulated hepatocytes, increased PINK1 and Parkin protein levels, and enhanced mitophagy, which subsequently inhibited NLRP3 and IL-1 expression, downregulated ROS production, and attenuated OA-induced hepatocyte injury ( In summary, enhanced mitophagy can reduce hepatic inflammation, OS, apoptosis, lipid accumulation, and steatosis, and inhibit the developmental process of MAFLD

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SdaA was reported to be the gene encoding either the structural gene of L-SD that catalyzes the deamination of serine to pyruvate or a positive activator of transcription [31]

[14] Peter Malliaras, Lower Limb Tendinopathy Course (London, 31.10.2016)