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Inflammatory mediators can induce production of prostaglandin E 2 (PGE 2 ) and NO, which in turn have the capacity to promote maturation and increase transport of melanosomes, to upregulate the expression of the tyrosinase gene, and to increase the complexity of the network of melanocytic dendrites

217 , e20190812 (2020)

This activation can change neuronal excitability and synchronization by modulating the function and expression of receptors, ultimately contributing to the development of epilepsy [5]

-Sitosterol has been reported to inhibit a variety of cyclin-CDK complexes, upregulate the cell cycle inhibitor p27, and cause cells to arrest in the G1 phase, among others (86, 87)