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Rather than treating ferroptosis as a uniform vulnerability, clinical translation will probably succeed where patients can be stratified by (1) the architecture of antioxidant defenses (for example, FSP1/GPX4 balance), (2) the size and distribution of cellular CoQ pools, (3) lipid composition and peroxidation-prone membranes and (4) oncogenic programs that remodel redox metabolism (such as NRF2KEAP1)
In a recent study involving the oral administration of liposomal glutathione, human subjects who took 500mg or 1000mg GSH daily had elevated stores of glutathione, and exhibited improvements in markers of immune function and oxidative stress [8]
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