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Description

In models of heart failure (HF), GLP-1 RAs have been observed to alter energy substrate utilization, alleviate myocardial and endothelial inflammation, mitigate oxidative stress, and antagonize myocardial remodeling [14]

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These include reducing oxidative stress caused by surgery and anesthetics, enhancing immunity, and reducing swelling, inflammation, and bruising

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FIGURE 4 Given the marked effects of HC on cell proliferation and migration, we next assessed whether HC treatment triggered apoptotic cell death

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TRICARE For Life and direct-care-only members are excluded as of August 31, 2025

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