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Given that L-carnitine uptake into skeletal muscle occurs slowly and is influenced by insulin availability and training status, chronic exposure to elevated plasma LC levels may eventually reduce the responsiveness of transport mechanisms or alter carnitine homeostasis

These agents enhance glucose-dependent insulin secretion from pancreatic beta cells, suppress inappropriate glucagon release, slow gastric emptying, and act on central nervous system appetite centers to promote satiety

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These agents exhibit anti-inflammatory, cardioprotective, and neuroprotective effects, suggesting a broader therapeutic potential